Cardiac hypertrophy review
[DOC File]Alglucosidase alfa - Pharmacy benefit management
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This medication was approved for use in patient’s ≥ 8 years old without cardiac hypertrophy suffering from late-onset Pompe disease.1 Pompe disease, also referred to as glycogen-storage disease type II or acid-maltase deficiency, is an autosomal recessive disease causing a deficiency in the lysosomal enzyme acid alpha-glucosidase (GAA).
[DOCX File]A novel immunomodulator agent: FTY720 targets NFAT ...
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G q stimulation of cardiac hypertrophy is well documented14, 15, and thus by inference we asked that FTY-720 may work through G i-dependent signaling to exert its anti-hypertrophic function. Firstly, we tested whether FTY-720 phosphorylation by sphingosine kinases (SPHKs) is a precondition for its anti-hypertrophic function.
[DOC File]CURRICULUM VITAE
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2004-2008 NIH Cardiac Contractility, Hypertrophy & Failure Study Section. 2004 VA Merit Review Subcommittee for Cardiovascular Studies-B. 2002-2004 NIH Cardiovascular and Renal Study Section, Ad hoc Member. 1997-Present NIH Ad hoc reviewer for individual grant proposals. 2000-2001 American Heart Association, National Peer Review Committee
[DOC File]PHS 398 (Rev. 9/04), Continuation Page
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, Kuppuswamy D: Integrin activation and focal complex formation in cardiac hypertrophy. J Biol Chem, 275(45): 35624-30, 2000. 4. Gramling-Babb P, Zile MR, Reeves ST: Preliminary report on high thoracic epidural analgesia (HTEA): Relationship between its therapeutic effect and myocardial blood flow as assessed by stress thallium distribution.
Role of miRNAs in cardiac fibrosis
Cardiac remodeling is characterised by cardiac fibrosis and hypertrophy. During cardiac fibrosis the TGF-β pathway is an important regulator of the ECM deposition. However, TGF-β has not seemed to be a possible therapeutic target because of its role during immune regulation and plaque formation.
[DOC File]Heart failure and PAH - Chest
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(91) Leinwand LA. Calcineurin inhibition and cardiac hypertrophy: a matter of balance. Proc Natl Acad Sci U S A 2001; 98(6):2947-2949. (92) Frey N, Olson EN. Cardiac hypertrophy: the good, the bad, and the ugly. Annu Rev Physiol 2003; 65:45-79.
[DOCX File]GeneDx
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The 2011 American College of Cardiology Foundation / American Heart Association (ACCF/AHA) guideline for the diagnosis and treatment of hypertrophic cardiomyopathy recommends genetic testing for HCM and other genetic causes of unexplained cardiac hypertrophy in patients with an atypical clinical presentation of HCM or when another genetic ...
[DOC File]Muscular System Review Sheet - Council Rock School District
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5. Cardiac Muscle – 6. Where is smooth muscle mainly found? 7. Skeletal muscles are mostly striated, what does this mean? 8. Muscle Flexor – 9. Muscle Extensor – 10. Example of flexor/extensor – 11. List 2 ways to build strong muscles. 12. What is protein? 13. List 4 good sources of protein. 14. What is a muscle strain and how do you ...
[DOC File]CAPRI GUI User Manual - Veterans Affairs
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Cindi Gawronski Jill Headen 10/12/2010 Answering ‘No’ to Section 5: Is there evidence of cardiac hypertrophy or dilatation? Allows user to reference the source and date of the test for patch 159. Cindi Gawronski N/A 10/28/2010 Changed wording in Introduction for patch 159.
DSpace
Cardiac hypertrophy is an increase in heart size due to either physiological or pathological reasons. Physiological inducers include exercise or pregnancy. For instance, athletes experience cardiac hypertrophy during their seasons of workouts. However, when they stop working out consistently, their hearts undergo cardiac regression, the ...
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