Digoxin toxicity symptoms in elderly

    • Digoxin Toxicity - What You Need to Know

      CHRONIC DIGOXIN TOXICITY. Introduction. Digoxin has a narrow therapeutic index and chronic intoxication can occur. It is usually seen in elderly patients with renal impairment, dehydration and co-morbidities. Digoxin immune FAB fragments are affective and should be given in these cases.

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    • [DOCX File]developinganaesthesia

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      digoxin toxicity is more like to produce bradyarrythmias than tachyarrythmias. acute digoxin OD in a non user will cause hyperkalemia. cardioversion is safe. in an elderly person who is digoxin toxic they are more likely to be hypokalemic. both hyper and hypokalemia exacerbate digoxin’s toxicity. 23.With regard to Isoniazid OD which is not true?

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    • [DOC File]TOXICOLOGY:

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      Digoxin toxicity resolution was maintained in these 7 patients (47%) at 4 h Fourteen patients (93%) were classified by investigators as having resolved their digoxin toxicity by 20 h. One patient was judged a non-responder after 24 h; this patient had continuing ECG abnormalities at the 30 day follow-up visit.

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    • [DOCX File]Attachment 2. Extract from the Clinical Evaluation Report ...

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      Monitor for cognitive dysfunction and depression in the elderly, as well as hallucinations and psychosis (more likely with higher doses). (These reactions appear to be related to the lipid solubility of this medication and its ability to cross the blood-brain barrier.)

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    • [DOCX File]Australian public assessment for Digoxin-specific antibody ...

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      Digoxin at a long-term dose greater than 125µg/day if eGFR < 30 ml/min/1.73m2 (risk of digoxin toxicity if plasma levels not measured). 2. Direct thrombin inhibitors (e.g. dabigatran) if eGFR ...

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    • [DOC File]Nursing Process Focus: Quinidine (Quinidex)

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      Digoxin is a cardiac glycoside used for the treatment of heart diseases such as certain types of arrhythmias and congestive heart failure. Digoxin has a narrow therapeutic range and digoxin toxicity can occur due to impaired renal function, drug interactions, alterations in plasma electrolytes (especially potassium and magnesium) and metabolic disturbances.

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