Dopamine pathways in schizophrenia
[DOCX File]Paper 2 Clinical: Studies – Carlsson et al (2000)
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some people with schizophrenia show dopamine levels within the normal range. ... pathways. glutamate acts as a . brake, signalling to GABA neurons to inhibit dopamine production ... PET scans show that people with schizophrenia given amphetamines show higher levels of dopamine release in the basal ganglia than age-matched controls and are more ...
[DOCX File]Psychology: Clinical – Content (AJW)
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Finally, although it is widely accepted that the majority of anti-psychotic drugs block dopamine receptors, to infer from this that dopamine hyper-activity is the major cause of schizophrenia is to oversimplify. It is now clear that the neurotransmitter systems interact and that the mapping of these cortical pathways is only just being explored.
[DOCX File]TITLE:
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the side effects of reducing dopamine levels are unpleasant, (e.g. extrapyramidal dysfunction, dysphoria and anhedonia) especially in the periods in between acute episodes, where people with schizophrenia may revert to more ‘normal’ levels of dopamine; arguably typical anti-psychotics (i.e. dopamine antagonists) bring the person down to ...
What is the role of dopamine in schizophrenia?
Schizophrenia Pathophysiology . Dopamine – increased dopamine activity in schizophrenia, affect many CNS pathways in brain. Subcortex – increased dopamine activity ( positive symptoms. Prefrontal – decreased dopamine activity ( negative symptoms. Evidence – increased dopamine receptors at autopsy, dopamine agonists worsen psychosis & vice-versa
[DOC File]10-30-07 Schizophrenia
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Two dopamine pathways are implicated in the etiology of schizophrenia: Mesolimbic System: This system is composed of the dopamine neurons from the ventral tegmental area that release dopamine to the nucleus accumbens.
[DOC File]Schizophrenia - Mercer University
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Schizophrenia is a chronic and disabling psychiatric illness and despite extensive research its molecular etiology remains unknown.1,2 The current dopamine hypothesis postulates excessive striatal dopamine transmission and reduced frontal dopamine stimulation underlying the pathophysiology of positive and negative symptoms, respectively.3,4 All currently approved treatments, the antipsychotics ...
[DOC File]Psychotic Disorders
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Neurotransmitters, Pathways, and Receptors. Positive Symptoms: Hyperactivity of Mesolimbic Tract. Negative Symptoms and Cognitive Impairment: Hyperactivity of the . Mesocortical Tract. Role of Other Dopamine Pathways. Role of Other Receptors. Psychological Theories. Social Theories. Interdisciplinary Treatment. Priority Care Issues
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