Dopamine uses and indications
Dopamine (Intropin): Side Effects, Dosages, Treatment, Interactions…
Dopamine produces nausea and vomiting by stimulation of the medullary chemoreceptor trigger zone (CTZ), and metoclopramide blocks stimulation of the CTZ by agents like l-dopa or apomorphine which are known to increase dopamine levels or to possess dopamine-like effects.
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Drug use also affects dopamine levels in the brain, impacting voluntary muscle movement in the rest of the body. An article in Current Drug Abuse Reviews showed that opioids, amphetamines, and cocaine all have the potential to cause ataxia because of their effects on dopamine levels.50 Additionally, withdrawal from frequent cannabis use and ...
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Altered dopamine signaling is known to be involved in schizophrenia. AKT1 can take one of three forms in a specific region of the gene implicated in susceptibility to schizophrenia: T/T, C/T, and C/C. Daily users of marijuana (green bars) with the C/C variant have a seven times higher risk of developing psychosis than infrequent marijuana users ...
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Indications include treatment of rumen atony, enhance gastric emptying, stimulation of peristalsis, emptying the bladders of large animals, diagnosis of myasthenia gravis in dogs and may be used to treat curare overdose. Side effects are cholinergic and include nausea, vomiting, diarrhea, drooling, sweating, lacrimation and bradycardia.
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Dopamine is a neurotransmitter normally produced by the body and it acts as a precursor to norepinephrine. Its effects ultimately depend on the dosage prescribed, as well as the patient’s condition. Dopamine given in low doses typically stimulates the receptors that produce vasodilation of the renal and mesenteric arteries.
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List the major therapeutic uses, adverse effects and drug interactions of NSAIDs. Explain the difference between COX-1 and COX-2 inhibitors. Know the names and primary adverse effects of the disease modifying anti-rheumatic drugs. Know the names, indications, mechanisms and primary adverse effects of the drugs used for gout.
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Describe two clinical uses (+ mechanism) for the synthetic prostaglandin Misoprostol (10) Inhibition of acid production by parietal cells => treatment of GERD, stomach ulcers etc. Increase in uterine muscle tone => used to induce labor, or in combination with mifeprostone, to terminate pregnancy.
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Describe two clinical uses (+ mechanism) for the synthetic prostaglandin Misoprostol (10) Which anti-diabetic drug group acts via the nuclear receptor PPAR ? Can these drugs be used for type I diabetes (explain!)? (10) What is the “Minimal Alveolar Concentration (MAC)”? How does the MAC relate to the lipophilicity of the drug? (10)
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Indications: 5. Normalization of pituitary function, especially as to Progesterone (Amphoteric) Irregular menses. PMS. Actions: Inhibits prolactin (yet useful as a galactagogue) Dopamine agonist. Dopamine agonist activity . Inhibits both basal and TSH-induced prolactin secretion. Binds to D2-receptor. Hormonal
Drugs found in the drug tray:
Increase dopamine levels (levodopa) Dopamine receptor agonists. MAO inhibitors. COMT inhibitors. Amantadine (Symmetrel) Decrease Acetylcholine activity. Anticholinergics. Drugs Used for PD. Levodopa (L-Dopa) Most reliable and effective drug used for PD. It is the biochemical precursor to dopamine- dopamine cannot cross BBB but levodopa can.
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