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[DOC File]Policy - Home Page | Boston Medical Center
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A salt wasting syndrome associated with cerebral disease. Transactions of the Association of American Physicians 1950; 63: 67-64 Pickard JD, Murray GD, Illingworth R, Shaw MD, Teasdale GM, Foy PM, Humphrey PR, Lang DA, Nelson R and Richards P et al. Effect of oral nimodipine on cerebral infarction and outcome after subarachnoid haemorrhage ...
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Cerebral edema can be severe, rarely life-threatening. Infants and young children most often present with seizures; older patients more often obtunded or comatose. Hypertension and bradycardia are inconsistent findings, especially in infants who may only have hypertension as ICP rises secondary to cerebral …
cerebral salt wasting syndrome

[DOC File]Fluid and Electrolyte Therapy in Children
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General Principles of Management. Corrected Na = measured Na + BSL – 5. 3. See below for specifics of management. Indications for hypertonic saline: coma, seizure, new onset profound LOC; not indicated if . asymptomatic. Dose: give . 25-100ml/hr (1-2ml/kg/hr) 3% saline. via CVL. Or . …
cerebral sodium wasting

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Neurocritical care should focus on neuromonitoring for delayed cerebral ischemia, management of hydrocephalus, seizures and intracranial hypertension, as well as of medical complications such as hyperglycemia, fever and anemia. ... so called 'cerebral salt wasting syndrome'.[159,160] Intravascular volume depletion and sodium loss may increase ...
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Cerebral Salt-Wasting Syndrome: Practice Essentials, Pathophysiolo…
Ascar A., Tarif N.: Cerebral salt wasting in a patient with head trauma: Management with saline hydration and fludrocortisone. Saudi J kidney dis. and transplant., 2007, 18(1): 95-99
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[DOC File]LEEDS TEACHING HOSPITALS TRUST
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c) Cerebral Salt Wasting is due to excess natriuresis. Low serum Na+. Low-normal serum osmolality Treat by sodium replacement. Coughing, restlessness and fighting the ventilator. These all raise the ICP and should be avoided by the use of adequate levels of sedation and, if required, paralysis (8). Patients should not be routinely paralyed.
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[DOC File]Harrison's Principles of Internal Medicine - 16th Ed
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Cerebral salt wasting (euvolemic or hypovolemic; moderate-high urine output) fludrocortisone 0.1-0.2mg PO/NGT BID. Normal saline to match urine output. If serum sodium < 130mEq/L, use 3% saline . Rate dependent upon degree of hyponatremia and volume status. Must be …
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[DOC File]Hyponatraemia in Primary Care - North Bristol NHS Trust
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Frequent monitoring of electrolytes is also important, since hyponatremia is a common finding up to two weeks following an SAH due to cerebral salt wasting or SIADH(Stern et al., 2006). Cerebral edema is also a common finding during this time.
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[DOC File]NEUROLOGICAL SUPPORT
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Cerebral salt wasting. Extra Renal loss; D&V. Burns. Bowel obstruction. Pancreatitis. Hypothyroid. Addison’s* SiADH. Admission may be required. Stop causative medications. Rehydrate and cause specific treatment. Admit if symptomatic or severe. Treat with fluid restriction. Urgent referral to endocrinology . Treatment is cause specific
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Cerebral salt wasting syndrom ( CSWS ) – nezvyklá ...
This "cerebral salt-wasting syndrome" clears over the course of 1 to 2 weeks and, in the setting of SAH, should not be treated with free-water restriction as this may increase the risk of stroke (see below). LABORATORY EVALUATION AND IMAGING (FIG. 349-14) The hallmark of aneurysmal rupture is blood in the cerebrospinal fluid (CSF).
cerebral salt wasting syndrome

Management of cerebral salt wasting