Pathophysiology of acute mi
[DOC File]Jordan University of Science & Technology
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etiology, pathophysiology, clinical significance, and management-discuss the nursing and collaborative management for clients with cardiac dysrhythmias. Chapter 18; pages 298-302. Chapter 18; pages 346-364, 368-370 6 V. Management of Clients with Acute Myocardial infarction (AMI)-review circulation to the cardiac muscle.
[DOC File]ARTERIAL OCCLUSIVE DISEASE
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PATHOPHYSIOLOGY OF ACUTE LL ISCHAEMIA (Essential Surgery 2nd Ed, pp 474, Fig 26.14) The acutely ischaemic lower limb is due to two main causes: 1) embolism, 2) thrombosis. Emboli most commonly arise from: 1) heart (atrial fibrillation, mitral stenosis, MI --> mural thrombus). The common sites of obstruction is at bifurcation (aortic, iliac ...
[DOC File]Guided Lecture Notes, Chapter 14, Nursing Management ...
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Summarize the pathophysiology, clinical manifestations, and treatment of myocardial infarction (refer to . PowerPoint slide 21) Discuss the rationale for using the term acute coronary syndrome (ACS). Explain the similarities and differences between the pathophysiology and manifestations of MI …
[DOC File]iiNet
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the diagnosis and differential of acute myocardial infarction & unstable angina . the pathophysiology of acute coronary syndromes (ACS) the pathophysiology of major cardiac complications after myocardial infarction . therapy in ACS that has been shown by randomised clinical trials (RCTs) to improve clinical outcome and survival
[DOC File]CV Pathophysiology
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NSTEMI – Partial obstruction, Positive enzymes (necrosis present) non Q-wave MI. STEMI – Complete occlusion of coronary artery, Q-wave MI (Q-waves show up later and are unpredictable so they are no longer used, but just so we know about them) Hemostasis. Primary hemostasis = Platelets plug up exposed collagen. Secondary hemostasis = Coag.
[DOC File]CV-Med5566
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recent acute MI (selection of patients is critical!!! Some can be harmed) extensive pulmonary emboli. severe deep vein thrombosis. thromboembolic stroke (tPAs. ... The use of activated protein C as a drug occurred as a result of a change in our understanding of the pathophysiology of sepsis, particularly the intricate interplay between ...
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12- PE pathophysiology ? 13- NSAID cause hemolytic anemia ? diclofenac . mefemanic acid . 14-NSAID effect on old age, side effect ? acute MI . 15-Descending colon blood supply ? inferior mesenteric artery . 16- Cut wound medial side of mid arm , 5 cm above the elbow , artery involved ?
[DOC File]Treatment of Anemia in Patients with Heart Disease
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Looking exclusively, then, at patients who had acute coronary syndrome or acute MI, there were 12 studies. Nine of these looked at outcomes across all hemoglobin or hematocrit levels after multi-varied adjustments and once again 8 of these 9 found a higher association or an association with higher mortality among patients who were transfused ...
[DOCX File]Pat Heyman - Family, Work, Play
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Describe the pathogenesis of MI including reversible/irreversible changes, major cause of death acutely, patterns of infarction (both distribution and mural portion), and reperfusion injury. ... Describe the pathophysiology acute pancreatitis. What are the major risk factors? ...
[DOCX File]EM Basic | Your Boot Camp Guide to Emergency Medicine
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Acute Coronary Syndrome- a spectrum of disease. Does NOT include low-risk chest pain (we admit low-risk chest pain to RULE OUT ACS) Unstable Angina. Pathophysiology. Fixed coronary stenosis that causes symptoms only when under stress. Can represent a patient with a “normal cath” with 30% stenosis. Definition
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