What is severe lv dysfunction

    • [DOC File]Valvular Heart Disease

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      Background Dilated Cardiomyopathy (DCM) is a heart muscle disease characterized by left ventricular (LV) or biventricular dilation and systolic dysfunction in the absence of either pressure or volume overload or coronary artery disease sufficient to explain the dysfunction (1–3).

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    • [DOCX File]Title: Cardiogenic shock with pulmonary edema (“cold and wet”)

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      In chronic severe AR, the left ventricle gradually enlarges while the patient remains asymptomatic. Symptoms usually develop after cardiomegaly and LV dysfunction have occurred. Dyspnoea is the principal complaint.

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    • Revascularization in Severe Left Ventricular Dysfunction: Does Myo…

      Severe LV dysfunction (anterior MI) Severe extensive ischemia. Mechanical complication of LVMI (acute MR, IVS rupture, tamponade) Outcomes: 30d mortality ~ 50%, accounts for 60% of 30d mortality following acute MI (Killip class IV = cardiogenic shock associated with 60-80% mortality)

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    • [DOCX File]Clinical Management of Dilated Cardiomyopathy

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      Given the association between reduced LV volumes and improved clinical outcome,6 albeit in patients with more severe LV systolic dysfunction, these data provide additional evidence for the use of CR exercise training in post-MI patients with preserved LVEF. …

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    • [DOCX File]Reverse left ventricular remodelling – effect of cardiac ...

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      with severe LV dysfunction caused by CAD. Therefore, in the absence of serious comorbid conditions, AVR is. indicated in virtually all symptomatic patients with severe AS. However, patients with severe LV dysfunction, particularly those with so-called low gradient AS, represent a difficult management decision (see above). AVR

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    • [DOC File]Intraaortic Balloon Pump (IABP)

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      (+) ganglionic blocking properties -> ↓PVR -> hypotension (severe if rapid IV or with severe LV dysfunction) CLASS I: SODIUM CHANNEL BLOCKERS CLASS IA: PROCAINAMIDE ANTI – ARRHYTHMIC DRUGS . PHARMACOKINETICS: Oral, IV, IM . N-acetylprocainamide (NAPA) -> major metabolite . Metabolism: hepatic . Elimination: renal . t½ = 3 to 4 hrs.

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    • [DOC File]Cardiology

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      -High risk PCI – LM disease, LV dysfunction, LM equivalent (40%), CHF -Bridge to Transplant – Not practical for long period time; usually need VAD -Mechanical complications of MI …

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    • [DOC File]FRACP- Cardiology answers

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      Limitations: severe mitral or aortic regurgitation and dilated cardiomyopathy may make interpretation difficult. Dobutamine echo is especially helpful in evaluating pts with LV dysfunction and severe aortic stenosis to quantify the extent of AS. Myocardial viability

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