White matter microvascular ischemic disease
[DOCX File]Update on small vessel diseases - University of Edinburgh
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This is whole posterior cerebral artery territory. He does not have other significant white matter microvascular ischemic changes or evidence of further previous infarcts.” The neurologist recommend CT angiogram to make sure that the patient did not have a dissection which was “low likelihood but patient is a heavy worker.”
[DOCX File]Viktor's Notes – Ischemic Stroke
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White matter pathology occurs in many CNS diseases including those of toxic/metabolic, developmental, ischemic, infectious/inflammatory, degenerative and neoplastic origins. Our ability to understand mechanisms of injury and distinguish reactive from pathological changes is hampered by the lack of tools for studying white matter.
[DOCX File]Neuropathology Division Strategic Plan 2015-2020
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Oct 31, 2014 · Schmidt R, Schmidt H, Haybaeck J, et al. Heterogeneity in age-related white matter changes. Acta Neuropathol 2011;122:171-185. Wardlaw JM, Smith EE, Biessels GJ, et al. Neuroimaging standards for research into small vessel disease and its contribution to ageing and neurodegeneration. Lancet Neurol 2013;12:822-838.
ischemic white matter disease - Neurology - MedHelp
Ischemic changes in the white matter can be seen by either 1H-MRS or DTI and the results are comparable.91 DTI allows measurements of fractional anisotropy and mean diffusivity.92 Quantification of DTI may be confounded by the heterogeneity of white matter regions with crossing fiber tracts, which can often be overcome with appropriate image ...
[DOCX File]Consensus statement for diagnosis of subcortical small ...
https://info.5y1.org/white-matter-microvascular-ischemic-disease_1_62478c.html
Generally, including in this review, CSVD is used to describe a series of imaging changes in white matter and subcortical grey matter, including recent small subcortical infarct, lacunes, white matter hyperintensities (WMH), prominent perivascular spaces (PVS), …
[DOC File]ACDIS
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A) CT < 24 h from stroke onset - loss of grey-white-matter differentiation in left frontal region and obscuration of caudate and lentiform nuclei; effacement of left frontal sulci. B) at 48 h infarct is well defined and exerts more mass effect.
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