ࡱ> }|} g^bjbj55 *__gV +++++????$c?"%/(WWWW2,$$$$$$$0')n$+22$++WW$ +W+W$ $ #$WW?a$$$0"%y$@*@*0$@*+$  $$ "%@* : August 2004 GP REGISTRAR INSTRUCTION SHEET OSCE Station 2 KNEE EXAMINATION The Scenario Your patient, Ms A Jones is a 30 year old PE teacher. She has a 2 year history of knee pain which has got worse in the last 2 months. The pain is at the front and lateral side of the knee. It is brought on by high impact activity with lots of knee flexion e.g. when she plays volley ball. It tends to ache after any energetic activity. There is no history of locking or instability The Task Please examine your patient, explaining what you are doing to the Assessor. You may ask further questions during your examination if you wish Notes The consultation will be stopped after 7-10 minutes allowing 5-7 minutes for feedback. July 2003 ASSESSORS INSTRUCTION SHEET OSCE STATION 2 KNEE EXAMINATION The aim of the station is to assess the GP Registrars ability to examine the knee joint. Patient 30 year old female PE teacher with patello-femoral pain syndrome If time permits, ask the registrar to demonstrate how he would examine for other abnormalities e.g. ligament injuries. It may be worth thinking of some scenarios to give appropriate clues. PLEASE NOTE IT IS YOUR RESPONSIBILITY TO RECRUIT A SUITABLE PATIENT Please brief the patient in his role before the day of the OSCE and give him the opportunity to clarify his role. Equipment needed (please arrange) Examination couch necessary During the consultation Take note of the registrars consultation skills in light of the Assessors feedback sheet (note items in bold print are of particular relevance for history given). Record significant behaviours, or their absence relevant for feedback. Stop the consultation after 7-10 minutes at your discretion, allowing 5-7 minutes for feedback. After the consultation 5-7 minutes will remain for feedback, demonstration and discussion. Please complete the checklist and hand to the registrar. Please note there is no patient instructions or feedback form. JULY 2003 ASSESSORS FEEDBACK SHEET OSCE STATION 2 KNEE EXAMINATION Ms A Jones AIM: To demonstrate the registrars ability to examine the knee joint. It is appreciated that in General Practice, the history will direct you to missing inappropriate parts of the examination. It is none the less important to be able to carry out all the examination. A = Completed satisfactorily B = Attempted/partially completed C = Not attempted NB: The items detailed below in bold print are of particular relevance for history given either for confirmation or exclusion purposes. ABCConsiderate, appropriate approaches to patient: introducing self clarifying position of pain consideration of pain explanation of examination and findings ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( )  Observation of patient in general (observes gait limp discomfort)  ( )  ( ) ( )  Observation of knee joint: bony landmarks muscle contours alignment AP and lateral muscle atrophy or hypertrophy swelling (effusion, synovial swelling bursitis tendinitis cysts erythema ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) Feel for: tenderness especially joint line swelling especially joint line temperature sensation ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( ) ( )Movement: active passive resisted movements of flexion, extension and hyperextension b) patellar movement crepitus instability ( ) ( ) ( ) ( ) ( ) ( )Tests for ligaments: lateral and medial collateral in extension + 20 flexion cruciate ligament injury drawer signs, Lachmans test, pivot shift test ( ) ( ) ( ) ( ) ( ) ( )Meniscal tests McMurrays test ( )( )( ) Not to be forgotten compares with normal side Referred pain from hip ( ) ( ) ( )  Examination completed in logical order ( ) ( ) ( )  Assessment of findings ( ) ( ) ( ) MAY 2003 ASSESSORS FEEDBACK SHEET GENERAL COMMENTS: PLEASE COMPLETE IMMEDIATELY AND HAND TO GP REGISTRAR AFTER THE FEEDBACK SESSION TOGETHER WITH ANY HANDOUTS GP REGISTRAR PLEASE SHOW THIS ASSESSMENT TO YOUR TRAINER, SO THAT YOU CAN DISCUSS IT FURTHER THEN KEEP IT IN YOUR LOG BOOK Handout Station 2 Notes on the assessment of the acutely injured knee Important points: The Knee - is the largest joint in the body - has no bony stability - is the most commonly injured joint in sport The Knee Ligaments - Are the most commonly injured structures and the ACL is the most commonly injured ligament - 70% of knee haemarthroses are due to an ACL tear - The average length of time from injury to diagnosis in an ACL tear is 21 months, the first doctor to see the injury only makes the diagnosis in 9% of cases. - In 90% of cases the diagnosis is based on the history. The History The history is vitally important and gives 70- 80% of the information to make a diagnosis - only 10- 20% additional/confirming information comes from the examination. Common situations to injure the knee are soccer, rugby, hockey and skiing in sport and also road traffic accidents. It is important to ask about the mechanism of injury but, unfortunately, in sport the precise mechanism of the injury often cannot be recalled due to the hurly-burly of the match. But attempt to find out if there was a pop or crack or snap from the knee as the player went down - was there an exquisite moment of pain? - Did the knee swell? - rapidly or later? - could the player carry on playing? or walk off the field? was he carried off? - did the knee give way? or lock? - what has happened since the injury? Pain - where is the site of pain? - laterally or medially -suggests ligaments and menisci - anteriorly - suggests patello-femoral pain. Swelling - a rapid onset <8hrs suggests haemarthrosis = ACL tear (70%) or osteochondral fracture - later onset - overnight = effusion ? meniscal lesion. Locking a knee that is (or was) stuck so it cant be straightened suggests a loose body - ? Mensiscal lesion - or an osteochondral fracture Pseudo-locking a knee that cannot be flexed with anterior pain suggests patellofemoral dysfunction. Giving way - reflex inhibition of quadriceps due to anterior knee pain causes partial giving way. - Complete collapse - esp. on rotation - i.e. an unstable knee = ACL lesion until proved otherwise. Examination Examination of the knee starts with observation as the patient enters the room, noting gait - limp - discomfort. Next looking at the patient - generally - then specifically at the knee and comparing the injured joint with the normal joint. Look for a) bony landmarks - muscle contours - alignment - AP and lateral - varus/valgus deformity - Q- angle b) muscle atrophy or hypertrophy c) swelling - (effusion - synovial swelling - bursitis - tendinitis - cysts) d) erythema - bruising Feel for a) swellings - effusion or synovial thickening - swollen bursae - joint line cysts - popliteal fossa cysts. b) tenderness - esp. joint line , ligaments and tendinous insertions. c) temperature d) muscle tone e) sensation f) pulses Move a) active - passive - resisted b) flexion - extension - hyperextension - flexibility of quads and hamstrings. c) patellar movement - crepitus - instability d) tests for the ligaments - lateral and medial collateral - in extension and 30 of flexion - instability in extension indicates major damage including tear of the posterior capsule, ACL and corresponding collateral ligament - NB important to abduct the hip to relax the IT tract when testing the LCL - instability at 30 isolates the collateral ligaments. Cruciate ligament tests - drawer signs, Lachmans test, pivot shift test. Tests of the cruciate ligaments are difficult to perform and unreliable, in the acute situation with pain and swelling the muscles (esp. hamstrings) will not relax sufficiently to allow the movement to occur for a positive sign. Lachmans and the pivot shift test require experience and a relaxed patient. Therefore the history is much more important than the physical signs. e) meniscal tests - McMurrays test - requires pain and a click Finally do not forget the possibility of referred pain from the hip and be aware of the possibility of generalized joint disease Investigations X-rays - AP and lateral - skyline view for the posterior border of the patella - tunnel view for the intercondylar notch Blood tests - ?WCC ? urate ? viscosity Aspiration - haemarthrosis - aspiration is therapeutic and diagnostic - presence of blood indicates arthroscopy for assessment of ACL effusion send fluid for microscopy and culture perform under asceptic conditions. Referral To fracture/trauma /acute knee clinic - for further investigation - arthroscopy, EUA or MRI scan - for follow up with rehabilitation For more detailed information on the assessment of knee injuries visit  HYPERLINK http://www.arc.org.uk/about_arth/med_reports/series3/pp/6340/6340.htm http://www.arc.org.uk/about_arth/med_reports/series3/pp/6340/6340.htm Anterior Cruciate Ligament rupture Large studies in the USA and UK have both demonstrated that this injury goes undetected for far too long. The average length of time from injury to diagnosis was 21 months in the USA and 22 months in the UK . The first doctor to see the patient after the injury made the diagnosis in only 9% of cases. It is likely that normal examination findings led doctors to ignore the history dont make the same mistake. Left undiagnosed what are the consequences of having an ACL deficient knee? That depends on what you want to do with your knees in the future. The ACL performs two important functions - as do all ligaments. Most obviously there is the provision of stability in movement of the joint. Less obvious is the proprioceptive function of the ligament. The ACL provides stability in the small amount of rotation that occurs in the knee joint. In the absence of the ACL the knee will tend to collapse when its owner turns. This was beautifully illustrated on national television in the FA cup final when Paul Gascoigne ruptured his ACL in a wild tackle. After attention from the man with the magic sponge he stood up, walked a little, jogged a little and then as he turned his knee gave way and he fell over. He was carried off on a stretcher and headed straight for the orthopaedic surgeon with his transfer value halved. Individuals who are not elite athletes may not require the attentions of a surgeon to reconstruct the ligament. Intensive rehabilitation focussing on hamstring function can to an extent provide sufficient rotatory stability for normal activities and some recreational, non-contact, uni-directional sport (cycling, swimming, rowing). The risk is, however, that activities like football, rugby and hockey are far too demanding and an unstable knee is likely to keep giving way with further damage to the joint occurring. Subsequent meniscal injury is one way that a missed ACL rupture comes to orthopaedic attention. In the long term it is known that an ACL deficient knee is likely to develop early osteoarthritis. What has not been shown, as yet, is whether surgical reconstruction prevents this. Long-term studies are underway and will answer this question in the near future. For the present all patients with suspected ACL rupture should be assessed by a knee specialist. Those that fail the rehabilitation regime and continue to have episodes of instability and those whose lifestyle or occupation depend on high level of knee function will probably be offered reconstruction. The current gold standard repair is a graft of the middle third of the patella tendon with a bone block at either end that is fixed into the lower femur and upper tibia. This is a highly skilled procedure and optimal function of the knee in the future requires optimal placement of the graft. As with many surgical procedures current thinking is that this should be done by a surgeon who is doing them on every list not once a month. Even with reconstruction intensive rehabilitation is required to restore proprioceptive function and it may well be the case that the elite athlete never regains the level of function required to regain their previous brilliance. (Hence the halving of Gascoignes transfer value) In summary for the elite athlete this is a career threatening injury. For the rest of us, and retired athletes, it is an injury that causes significant limitation of activity, can lead to further joint injury and early joint destruction. Dont miss one. Posterior Cruciate Ligament Rupture The posterior cruciate ligament (PCL) is less commonly injured (4% of acute knee injuries) and the consequences of rupture are less spectacular. The mechanism of injury may be acute hyperextension or, more commonly, a forceful direct blow to the anterior tibia with the knee in flexion. Common situations are ski-ing and hitting the dashboard in front impact vehicle collision in addition to the hurly-burly of contact sport. PCL injury may accompany other knee injuries in severe trauma. This injury is difficult to diagnose because it can be asymptomatic once the symptoms of the acute insult to the knee have settled. Clearly the PCL is less important for knee stability than the ACL. The patient may complain of vague knee pain, sometimes patello-femoral pain due to the increased pressure on the patello-femoral joint with posterior displacement of the tibia. Sometimes the patient experiences giving way but apart from in skiing where the PCL is vital for maintaining an aerodynamic tuck position the injury can cause surprisingly few symptoms. Management is therefore usually conservative with the focus being on intensive rehabilitation especially to quadriceps function, the quads providing a degree anterior stability to the knee. As with ACL injuries the late consequences of PCL rupture appear to be early degenerative change. Medial Collateral Ligament Injury Medial collateral ligament injury comprises 29% of acute knee injuries. The history is of an acute valgus force to the partially flexed knee. Again common situations are contact sports and downhill skiing. MCL injuries are graded according to severity. A grade 1 injury may cause medial knee pain, tenderness over the medial femoral condyle (this differentiates the injury from a medial meniscus injury which causes joint line tenderness) and pain but no joint laxity when the knee is subjected to valgus stress at 30( of flexion by the examiner. A grade 2 injury will have some swelling over the medial condyle and exhibit pain and some laxity when subjected to the same valgus stress. A grade 3 lesion is a complete tear and the patient may be aware of a wobbly knee lateral instability. There may little pain and swelling but valgus stress at 30( reveals gross laxity. The outlook for MCL injuries is good in contrast to the ACL injuries. Conservative treatment with early mobilisation and intensive rehabilitation produces just as good results as surgical reconstruction whatever the severity of the injury. Protection of the joint in a brace that allows mobilisation may required for the more severe injury with instability. Early mobilisation prevents joint stiffness and loss of muscle power that can occur rapidly in knee injuries. Lateral Collateral Ligament Injury Lateral collateral ligament injury is significantly less common than MCL injury (2% of acute knee injuries). This presumably relates to the varus impact required to create the injury being less common in sport - the other leg getting in the way!? The recognition grading and management are the same as for MCL injury. For more information on ligamentous injuries of the knee refer to: Br J Sports Med 2000; 34:395-400  HYPERLINK "http://www.bjsportmed.com/search.dtl" www.bjsportmed.com/search.dtl Further Reading: Two more cases of acute knee injury 1) Sally, a 25 year old hockey player relates how one month ago in a match she was tackled when running at speed. She remembers falling and a moment of severe pain in her right knee. She describes feeling and hearing a loud crack. She was carried to the touchline and her knee was bandaged up by a St John's Ambulance man. After the match her friend took her to Casualty as the knee had become very swollen. She had an X-ray, which was normal, and was then put in an enormous bandage. The swelling seemed to settle down after a week of rest and she resumed walking without difficulties. Last night she went to her first training session and in the first few minutes the knee gave way as she went into a tackle and she fell to the ground. Overnight the knee has swollen again. This is the typical late presentation of a common acute knee injury. I really want you to remember this history. This is an injury that, sadly, goes undiagnosed for long periods of time during which time irreversible damage is occurring to the joint. The important features of this history are: This was a dynamic injury it occurred whilst running at speed There was a moment of exquisite pain accompanied by a crack or snap The athlete was unable to continue There was a rapid onset of swelling The X ray was normal There was no follow up at the A&E department! One month later the knee gives way After the episode of giving way the knee has swollen again more slowly Irrespective of any other information you may discover, this patient has ruptured her anterior cruciate ligament (ACL) unless and until the ligament has been visualised at arthroscopy by a knee specialist and pronounced intact. If you wish, you may try to elicit the fact that the knee gives way on rotation but otherwise the history is complete. Examining the knee to demonstrate the ACL deficiency requires some skill in performing some manoeuvres you might just remember from medical school orthopaedics. These include Lachmans test and the pivot shift test. The only point in mentioning them here is to say that in unskilled hands they are unreliable and I would consider most general practitioners including myself to have unskilled hands as far as these tests are concerned. You really need to be using them almost every day to develop the necessary feel for the joint. So when you come to examine this patients knee you may find a number of signs or nothing at all. If you had seen the knee shortly after the injury in A&E you would have found a tense swollen joint that was far too sore to do anything with. You may have aspirated the joint to demonstrate the haemarthosis that is suggested by the rapid onset of swelling in the history. In surgery the morning after the second incident the longer onset of swelling (overnight) suggests an effusion. In between these two episodes apart from loss of quadriceps bulk and power (which occurs rapidly in any painful insult to the knee) you may be unable to demonstrate any significant signs. It is absolutely vital that having examined the knee and found little of note that you should not ignore a history like this one and fail to refer the patient to a knee specialist. The posterior cruciate ligament is less commonly injured and the consequences of rupture are less spectacular. 2) A 35 year old man presents in the Monday morning surgery with an acutely painful and swollen left knee. He describes playing football yesterday morning. As he was about to cross the ball into the goal from the left corner he felt severe pain on the inside of his left knee. He collapsed to the ground. For a while the knee felt to be stuck in one position and he was unable to stand up. Somehow the knee unlocked itself and he was able to stand up and hobble around. Later in the day the knee started to swell and over night has swollen considerably. This is another common acute knee injury presenting the morning after the match. The important features of this history are: The injury occurred when the athlete rotated on a flexed knee Severe medial knee pain The knee locked for a length of time The swelling occurred slowly overnight There is a saying amongst doctors involved in sport that football is not so much a sport as a knee disease. Although this injury can occur in many activities, sporting or otherwise, football seems to present the lions share. A locked knee suggests a loose body. A knee that locks (not pseudo-locking as can occur in patello-femoral joint problems) needs a look inside to identify and deal with the loose body. The loose body in this situation is highly likely to be a piece of torn medial meniscus that is flapping in the breeze. The swelling developing slowly is an effusion. When you examine this knee you may find little more than the effusion. Just as with the acute ACL injury an acutely inflamed joint is a thing that its owner tends not to want anyone to play with. The musculature around the joint will be holding the joint very preciously. Quadriceps wasting occurs surprisingly quickly but perhaps not this quickly. There may be medial joint line tenderness over the damaged meniscus. The provocative tests of meniscal integrity again require skill and a relaxed patient and a quiet joint. So to repeat the message the history provides the diagnosis the examination findings may confirm the diagnosis but not invalidate it. This patient requires referral to the nearest acute knee clinic. If attended to early some meniscal injuries can be repaired with good results. The outer rim of the meniscus has a blood supply which will allow healing to occur if the tear is sutured. Other, more central lesions require removal of the damaged segment and tidying up of whats left behind. The days of completely removing damaged menisci are thankfully long past. We now know how important the menisci are in joint function. There are unfortunately many former athletes around who had injured menisci removed over 20 years ago and who are now reaping the rewards with early osteoarthritis of the knee. .?PQ    , - K \ l !(03567PQOQRSüõüõõ맵ӛӧͧӛӛӛӛ h6D5CJ h6D6 h6D5h6D56CJ h6D6CJ h6D6CJh6D56>*CJ h6DCJ h6DCJh6DOJQJ]h6D5>*CJh6D h6D5CJh6DCJOJQJ=./>?Q^_ 8 z {    , - $a$gdCJ^- K L J K m n I M & F & FM!"#23Pr $$Ifa$$If 0^`0 & FGXt $$Ifa$ & F$If$If & F$If[kd$$IflFJf!#vF@    4 lawqg & F$If$If~kdu$$IflrhJf!#lvF4 la $$Ifa$ <=BCDIJOPQk~kd$$IflrhJf!#lvF4 la $$Ifa$$If$If QRSn"',1238=BGHINSX] $$Ifa$ & F$If$If & F$If]^_`jwmg]]]] & F$If$If & F$If~kd$$IflrhJf!#lvF4 la $$Ifa$ S^_`j`XZcenopq<=>jkpG$,ŽŬh6D5>*OJQJh6DOJQJ h6D>*CJh6D56>*CJh6D56>*h6D h6D5h6DCJOJQJh6DCJOJQJh6D5CJ\ h6DCJ\ h6DCJ h6D5CJ< $$Ifa$ `vpfp]]]]] $$Ifa$ & F$If$If & F$If~kdX$$IflrhJf!#lvF4 la wmg$If & F$If~kd$$IflrhJf!#lvF4 la $$Ifa$ NOTYZ_dejopm~kd$$IflrhJf!#lvF4 la $$Ifa$ & F$If pqg~kd;$$IflrhJf!#lvF4 la $$Ifa$$If & F$If *wqgqq & F$If$If~kd$$IflrhJf!#lvF4 la $$Ifa$ *+0167<=>?@Wwqgqq & F$If$If~kd}$$IflrhJf!#lvF4 la $$Ifa$ WX]^cdijklmnowqq$If~kd$$IflrhJf!#lvF4 la $$Ifa$ opqrs~~~~tttttttt 0^`0~kd$$IflrhJf!#lvF4 la  ^` 0^`0FG"#Ql^p^p$a$ ^`".KG!S!F"Q"=#G#1$6$$%$%<&M&((U*_*.+/+++++++++999>?AAhBiBVDyDEŸzz jh6DOJQJh6D6CJOJQJ\]h6D0JOJQJj`h6DOJQJUjh6DOJQJUh6D>*OJQJmH sH h6DOJQJmH sH h6D>*OJQJ h6DCJh6DOJQJh6Dh6D5OJQJh6D5>*OJQJ.E!"./KQRA G!S!T!!!E"F"$a$$^a$$a$ $ L^$ `L $$ L^$ `La$F"Q"R"""%#<#=#G#H### $$&$0$1$6$7$W$$$'?(A((((L)u)u)v)T*U*_*`****++++-- 13r7s7889999=>>>>>>??BUDVDyDzDEEEEuFvFwFFFFIIJJJJ#KkKK$a$E FFF!F"FTFUFVFsFtFvFwFFzR/Sg^ؿh6D5CJOJQJh6DOJQJh6Dh6D0JOJQJjh6DOJQJUjh6DOJQJUh6D0JOJQJh6DOJQJh6D6OJQJKKKKLfLgLKMMyRzR/S0SSSUUVVKVVVVVVW5Y6Y[f^f^g^(. 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