Pharmacology of dopamine
[DOC File]“DMSH Pharmacological Practice Guidelines for the ...
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Associate Professor of Pharmacology & Physiology . UMDNJ- New Jersey Medical School . Newark, New Jersey “Pre-clinical Development of a New Class of Atypical Dopamine Receptor Agonists to Treat Dyskinesias: WEDNESDAY, January 18, 2012. 10:30 a.m. - 12:00 Noon* MSB – Room B-610 *Followed by: Brown Bag Lunch Meeting with NJMS Residents
[DOC File]Autonomic Nervous System Pharmacology
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To discuss the pharmacology of the sympathetic and parasympathetic nervous system can be overwhelming. The number of drugs that influence these systems is immense, and you will be hard pressed to find drugs that do not either directly or indirectly interact with the SNS/PNS. ... α receptors, β receptors and dopamine receptors (sympathetic ...
[DOC File]Pharmacology
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A. Dopamine Agents. 1. Carbidopa / Levodopa (Sinemet): Carbidopa inhibits peripheral use of dopamine making more available to the brain. Levodopa is converted to dopamine. 2. Levodopa (Levopa) 3. Amantadine (Symmetrel): increases the release of dopamine. 4. Parlodel: increases dopamine in the brain. B. Cholinergic Blocking Agents. 1. Examples ...
[DOC File]Pharmacology—Antipsychotic Drug Therapy
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Dopamine Hypothesis. The dopamine hypothesis . suggests that excessive dopaminergic activity underlies schizophrenia includes the following: Drugs that increase dopaminergic activity either aggravate existing schizophrenia or induce psychosis. ... Pharmacology—Antipsychotic Drug Therapy ...
[DOC File]Pharmacology - Weebly
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A. Dopamine Agents. 1. Carbidopa / Levodopa (Sinemet): Carbidopa inhibits peripheral use of dopamine making more available to the brain. Levodopa is converted to dopamine. 2. Levodopa (Levopa) 3. Amantadine (Symmetrel): increases the release of dopamine. 4. Parlodel: increases dopamine in the brain. B. Cholinergic Blocking Agents. 1. Examples ...
[DOCX File]Dopamine - Safer Care Victoria
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Mechanism of action/pharmacology. Dopamine is an immediate catecholamine precursor of noradrenaline that directly stimulates alpha, beta and peripheral dopaminergic receptors in a dose-dependent manner, as well as acting indirectly by releasing endogenous noradrenaline from storage sites in sympathetic nerve endings.
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