Salt wasting brain

    • Cerebral salt-wasting syndrome - Wikipedia

      Ascar A., Tarif N.: Cerebral salt wasting in a patient with head trauma: Management with saline hydration and fludrocortisone. Saudi J kidney dis. and transplant., 2007, 18(1): 95-99

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    • [DOCX File]www.oapublishinglondon.com

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      Cerebral salt wasting (euvolemic or hypovolemic; moderate-high urine output) fludrocortisone 0.1-0.2mg PO/NGT BID. Normal saline to match urine output. If serum sodium < 130mEq/L . NaCl 1g tabs; 1-2 tabs PO/NGT BID-QID. OR. 3% saline infusion. Rate dependent upon degree of hyponatremia and volume status. Must be given through central line or ...

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    • [DOC File]Neurological assessment of head injuries using the Glasgow ...

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      Structure and function of the beta 2 subunit of brain sodium channels, a transmembrane glycoprotein with a CAM motif. ... S. S., et al., 1996. Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1. ... 2002. Monogenic tubular salt and acid transporter disorders. J ...

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    • [DOC File]Harrison's Principles of Internal Medicine - 16th Ed

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      Cerebral salt wasting: pathophysiology, diagnosis, and treatment. Neurosurg Clin N Am. 2010;21(2):339-52. 4.Kirkman MA, Albert AF, Ibrahim A, Doberenz D. Hyponatremia and brain injury: historical and contemporary perspectives. Neurocritical care. 2013;18(3):406-16. 5.John CA, Day MW. Central neurogenic diabetes insipidus, syndrome of ...

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    • [DOC File]Policy

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      Cerebral Salt Wasting Syndrome. CSW means that the body is excreting salt in the urine. Salt has a high osmotic action so carries water with it. This results in a large diuresis in addition to a low serum sodium. The patient will be dehydrated, confused, hypotensive and deteriorating.

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    • Cerebral salt wasting syndrom ( CSWS ) – nezvyklá ...

      This "cerebral salt-wasting syndrome" clears over the course of 1 to 2 weeks and, in the setting of SAH, should not be treated with free-water restriction as this may increase the risk of stroke (see below). LABORATORY EVALUATION AND IMAGING (FIG. 349-14) The hallmark of aneurysmal rupture is blood in the cerebrospinal fluid (CSF).

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