What is cardiac hypertrophy

    • [PDF File]PDF Potential Markers in Cardiac Hypertrophy

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      POTENTIAL MARKERS IN CARDIAC HYPERTROPHY? POTENTIAL MARKERS IN CARDIAC HYPERTROPHY? Bartosz Malinowski 1,2, Gabriele Fulgheri 1, Michal Wicinski 2, Elzbieta Grzesk 2, Grazyna Odrowaz‐Sypniewska 1, Grzegorz Grześk 2, Nasser Darwish2 1Department of Laboratory Medicine, Collegium Medicum, Nicolaus Copernicus University, Bydgoszcz, Poland

    • Cardiac hypertrophy elevates serum levels of fibroblast ...

      Cardiac hypertrophy elevates serum levels of fibroblast growth factor 23 see commentary on page 7 Isao Matsui1, Tatsufumi Oka1, Yasuo Kusunoki2, Daisuke Mori1, Nobuhiro Hashimoto1, Ayumi Matsumoto1, Karin Shimada1, Satoshi Yamaguchi1, Keiichi Kubota1, Sayoko Yonemoto1, Tomoaki Higo3, Yusuke Sakaguchi4, Yoshitsugu Takabatake1, Takayuki Hamano4 and Yoshitaka Isaka1

    • Soluble epoxide hydrolase inhibitor, TUPS, protects ...

      Cardiac hypertrophy can be broadly defined as an increase in heart mass in response to an increase in biomechanical stress. The growth of heart is characterized by the growth of indi-vidual cardiomyocytes rather than an increase in cell number (Vakili et al., 2001). Cardiac hypertrophy is traditionally con-

    • [PDF File]PDF cardiac hypertrophy, fibrosis, and diastolic Fibroblast ...

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      pathogenesis of cardiac disease, including cardiac hypertrophy and fibrosis, there is increasing evidence that cardiac fibroblasts (CFs), the most abundant cell type in the mammalian heart, could also play an important role in mediating cardiac hypertrophy and remodeling (16-18). However, the role of ROCK2 in

    • [PDF File]PDF Pregnancy-Associated Cardiac Hypertrophy in Corin-Deficient ...

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      cardiac hypertrophy, and preeclampsia.20-24 Decreased corin levels have been reported in the uterus and serum of pre-eclamptic women.22,25-28 It remains unknown if and to what extent corin defects contribute to HDP-associated cardiac changes. HDP and subsequent cardiovascular disease (CVD)

    • Hypertension, Cardiac Hypertrophy, and Impaired Vascular ...

      cardiac hypertrophy, which may be mediated, in part, by increased superoxide. Keywords Aryl hydrocarbon receptor Cardiac hypertrophy Endothelial dysfunction Hypertension Superoxide TCDD Introduction 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) is a ubiqui-tous toxicant that persists in the environment and is generally resistant to degradation.

    • Mechanisms of Cardiac Hypertrophy

      Mechanisms of Cardiac Hypertrophy Giulio Selvetella, MDa, Giuseppe Lembo, MD, PhDa,b,* aIRCCS Neuromedicine, Pozzilli, Italy b''La Sapienza'' University of Rome, Rome, Italy Cardiac hypertrophy is a response of myocardium to various physiologic and pathologic stimuli, me-

    • [PDF File]PDF Cardiac Hypertrophy: A Review on Pathogenesis and Treatment

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      Cardiac hypertrophy has been considered as an important risk factor for cardiac morbidity and mortality whose prevalence has increased during the last few decades. Cardiac hypertrophy, a disease associated with the myocardium, is characterized

    • [PDF File]PDF Eccentric and concentric cardiac hypertrophy induced by ...

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      eccentric cardiac hypertrophy induced by exercise training. We also describe the involvement of the angiotensin II type I (AT1) receptor and of the microRNAs (miRs) in different forms of cardiac hypertrophy. Concentric cardiac hypertrophy induced by resistance training In static or isometric physical exercise (e.g., weight lifting,

    • [PDF File]PDF Cardiac hypertrophy and thyroid hormone signaling

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      cardiac hypertrophy to a normal cardiac configuration. In addition, less well recognized effects of thyroid hormones are exerted on the cardiac vascular system with a promo-tion of angiogenesis [8, 9]. Increased thyroid hormone action leads to a decrease of the tone of arterial vascular smooth muscle and a markedly decreased cardiac afterload ...

    • [PDF File]PDF Tenascin-C in Cardiac Hypertrophy and Fibrosis

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      EDITORIAL COMMENT Tenascin-C in Cardiac Hypertrophy and Fibrosis Friend or Foe?* Woo Jin Park, PHD, a,b Dongtak Jeong, PHD, Jae Gyun Oh, PHDa T enascin-C (TNC) is a large, hexameric, extracellular matrix glycoprotein.

    • Cardiac Hypertrophy: A Risk Factor for QT-Prolongation and ...

      Cardiac Hypertrophy: A Risk Factor for QT-Prolongation and Cardiac Sudden Death Y. J AMES KANG Departments of Medicine, Pharmacology and Toxicology, University of Louisville School of Medicine, Louisville, Kentucky 40292, USA ABSTRACT Cardiac hypertrophy was viewed as a compensatory response to hemodynamic stress.

    • [PDF File]PDF Cardiac Nuclear High-Mobility Group Box 1 Ameliorates ...

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      Cardiac hypertrophy was induced in 10- to 12-week-old mice with cardiac-specificover-expression of HMGB1 (HMGB1-Tg) and their wild-type (WT) littermates by chronic infusion of Ang II (1.5 mg/kg/day) or saline as previously described(23).

    • [PDF File]PDF Raf-mediated cardiac hypertrophy in adult Drosophila

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      Cardiac hypertrophy is a common condition that is triggered by environmental or genetic cues in which the heart muscle enlarges because of an increase in the size of cardiomyocytes. If untreated ...

    • [PDF File]PDF Cardiac hypertrophy is with

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      c-mycexpression in cardiac myocytecultures. Thesefrdings show that the induction of cardiac myocyte hypertrophy is associated with enhanced expression of the c-myc gene and suggest that hormonally induced cell hypertrophy and cell division share commonmechanistic pathways. Tissues grow by two processes: an increase in cell size

    • [PDF File]PDF Is Cardiac Hypertrophy Good or Bad? - JACC Imaging

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      Is Cardiac Hypertrophy Good or Bad? The Answer, Of Course, Is Yes* Blase A. Carabello, MD I nthisissueofiJACC,Petrovetal.(1) examined patterns of hypertrophy in patients with aortic stenosis. They found that women had"adap-tive" hypertrophy more often than men, but when women had "maladaptive" hypertrophy, their mor-

    • [PDF File]PDF VA Form 21-0960A-4 Non-Ischemic Heart Disease (Including ...

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      NOTE: For coronary artery disease, myocardial infarction, or hypertensive disease, complete VA Form 21-0960A-1, Ischemic Heart Disease Disability Benefits Questionnaire. SECTION I - DIAGNOSIS. HEART CONDITIONS (INCLUDING ISCHEMIC AND NON-ISCHEMIC HEART DISEASE, ARRHYTHMIAS, VALVULAR DISEASE AND CARDIAC SURGERY)

    • Differential cardiac hypertrophy and signaling pathways in ...

      Cardiac hypertrophy is an independent risk factor for major cardiac events (6, 47). Mechanical overload is the most impor-tant contributor to cardiac hypertrophy (47); it can be generally differentiated into pressure overload and volume overload, causing morphologically distinct types of cardiac remodeling.

    • Cardiac Hypertrophy: Useful Adaptation or Pathologic Process?

      namic factors and the cardiac hypertrophy of normal human growth may be gained by examining the rela- tionship between ventricular wall stress and the pattern of cardiac hypertrophy in patients with chronic pres- sure-overload or volume-overload of the left ventricle.

    • [PDF File]PDF A Redox-Dependent Pathway for Regulating Class II HDACs and ...

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      A Redox-Dependent Pathway for Regulating Class II HDACs and Cardiac Hypertrophy Tetsuro Ago,1 Tong Liu,2 Peiyong Zhai,1 Wei Chen,2 Hong Li,2 Jeffery D. Molkentin,3 Stephen F. Vatner,1 and Junichi Sadoshima1,* 1Department of Cell Biology and Molecular Medicine, Cardiovascular Research Institute, University of Medicine and

    • [PDF File]PDF hypertrophy-independent cardiac dysfunction

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      cardiac hypertrophy, which were largely normalized by preventing the recruitment of PI3K by bAR kinase 1 to ligand-activated receptors. Thus stress-induced activation of pathogenic signaling pathways, not the duration of stress or the hypertrophic growth per se, is the molecular trigger of cardiac dysfunction. Introduction

    • Targeting TMBIM1 Alleviates Pathological Cardiac Hypertrophy

      a MVB regulator in the progression of pathological cardiac hypertrophy, as well as the role of vesicle trafficking in signaling regulation dur ing cardiac hypertrophy. Moreover, targeting TMBIM1 could be a novel therapeutic strategy for treating cardiac hypertrophy and heart failure.

    • Glucose Metabolism in Cardiac Hypertrophy and Heart Failure

      Glucose Metabolism in Cardiac Hypertrophy and Heart Failure Diem H. Tran PhD; Zhao V. Wang, PhD H eart failure is one of the leading causes of death worldwide and has been singled out as an emerging epidemic.1,2 With a 5-year survival rate of 50%, heart failure

    • [PDF File]PDF ACVIM Fact Sheet: Hypertrophic Cardiomyopathy in Cats Overview

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      hypertrophy is mild and focal, the cat may remain symptom-free for all his life. However, if the hypertrophy is severe, the ventricle will have a hard time distending, which leads to increased intra-cardiac pressure and congestive heart failure (CHF) with fluid build-up in or around the lungs.

    • [PDF File]PDF Histone deacetylase (HDAC) inhibitors attenuate cardiac ...

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      afterload-induced cardiac hypertrophy/failure triggered by tight banding of the aorta and that this load-induced autophagic re-sponse is maladaptive, contributing to disease pathogenesis (13). To examine further the role of cardiomyocyte autophagy in pathological cardiac remodeling, we used a model of moderate

    • [PDF File]PDF The Role of Pontin in Stress-induced Cardiac Hypertrophy

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      CARDIAC HYPERTROPHY A THESIS SUBMITTED TO THE UNIVERSITY OF MANCHESTER FOR THE DEGREE OF DOCTOR OF PHILOSOPHY IN THE FACULTY OF BIOLOGY, MEDICINE AND HEALTH 2018 Thuy Anh Bui School of Medical Sciences Division of Cardiovascular Sciences . 1

    • Cardiac Hypertrophy in Obstructive Sleep Apnea Syndrome

      ular hypertrophy in OSAS has not been established. We hypothesized that the presence of cardiac hy­ pertrophy was related to the severity of OSAS, an

    • [PDF File]PDF Resistance Training and Cardiac Hypertrophy

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      Resistance Training and Cardiac Hypertrophy Unravelling the Training Effect Mark J. Haykowsky,1,2 Rudolph Dressendorfer, 1 Dylan Taylor,2 Sandra Mandic 1 and Dennis Humen3 1 Faculty of Rehabilitation Medicine, University of Alberta, Edmonton, Alberta, Canada

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